A quick review of this, and similar, study and the literature indicates that muscle glycogen stores are reduced by exertion. In a controlled study, a high-intensity interval training program that included a weight with 10 percent of maximal resting oxygen uptake (max) resulted in increased glycogen stores compared to a low-intensity interval training program which included 10 percent of maximal oxygen uptake. Furthermore, during the training period, muscle glycogen stores were lower between high intensity interval training and a low intensity interval training program. The authors concluded,
Our findings suggest that prolonged and sustained high energy (MR) exercise enhances muscle glycogen storage and promotes insulin sensitivity and blood glucose regulation (16), and suggest that high-intensity interval training may be used as a treatment for metabolic syndrome (ICS), diabetes, and chronic fatigue syndrome. In patients presenting with ICS, high intensity interval training may facilitate muscle recruitment that may help maintain muscle volume during exercise and facilitate body composition changes after exercise. However, prolonged high-intensity interval training may result in low-grade shortness of breath and poor muscle health, which may contribute to increased insulin sensitivity and blood glucose regulation. High-intensity interval training may promote muscle glycogen storage via increased insulin sensitivity, increased blood glucose regulation, and improved overall blood volume (17).
This study appears in the International Journal of Obesity.
References
- J.S. Hocha, A. K. Rupp, K. S. Kramnik, G. P. Albertson, M. K. Wijns, T. A. M. Flemming, B. A. Rs, P. Y. van der Laan, M. Lecchi, G. A. Hebert, P. J. Lehr, M. J. P. Golles, M. L. A. Hebert, P. B. Mihanovic, J. F. Zenger, S. A. Reiss, J. V. Voswold, R. G. van der Sijnl, A. S. Jensson, D. C. Weijers, G. H. Bijmel, D. A. H. Van Pelt and T. B. Kosten. 2014. High-intensity interval training for weight training in patients with hypovolemic (VO2max) hypocalcephaly. Eur. J. Physiol. 295 735740.
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